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Alexander, F. (1954). Some observations on general anaesthesia in ponies. J Comp Pathol, 64(1), 20–25.
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Alexander, F., & Chowdhury, A. K. (1958). Enzymes in the ileal juice of the horse. Nature, 181(4603), 190.
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Alexander, F., & Davies, M. E. (1963). Production and fermentation of lactate by bacteria in the alimentary canal of the horse and pig. J Comp Pathol, 73, 1–8.
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Alexander, F. (1952). Some functions of the large intestine of the horse. Q J Exp Physiol Cogn Med Sci, 37(4), 205–214.
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Alexander, F., Macpherson, M. J. D., & Oxford, A. E. (1952). Fermentative activities of some members of the normal coccal flora of the horse's large intestine. J Comp Pathol, 62(4), 252–259.
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Alexander, F. (1978). The effect of some anti-diarrhoeal drugs on intestinal transit and faecal excretion of water and electrolytes in the horse. Equine Vet J, 10(4), 229–234.
Abstract: The effect of morphine, Tinct. opii, loperamide, pethidine and atropine on intestinal transit and the faecal and urinary excretion of water and electrolytes was studied in ponies. The rate of passage of a particulate marker was slowed by morphine, hastened then slowed by loperamide and Tinct. opii, and hastened by atropine. The liquid marker was slowed by Tinct. opii and hastened then slowed by the other drugs. Only loperamide decreased the faecal sodium excretion. This drug also decreased faecal water and weight; it appeared worthy of clinical trial in diarrhoea. Tinct. opii decreased by morphine, pethidine and atropine increased faecal water.
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Alexander, F. (1966). A study of parotid salivation in the horse. J Physiol, 184(3), 646–656.
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Alexander, F., Davies, M. E., & Muir, A. R. (1970). Bacteriophage-like particles in the large intestine of the horse. Res Vet Sci, 11(6), 592–593.
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Alexander, F., & Davies, M. E. (1969). Studies on vitamin B12 in the horse. Br. Vet. J., 125(4), 169–176.
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Alexander, F. (1977). The effect of diuretics on the faecal excretion of water and electrolytes in horses. Br J Pharmacol, 60(4), 589–593.
Abstract: 1. The effect on plasma, urinary and faecal electrolytes of frusemide and hydrochlorthiazide was measured in ponies, mean weight 180 kg. 2. The rapid loss in urine of large quantities of sodium had only a small effect on plasma sodium concentration. 3. Faecal sodium excretion was increased substantially after the administration of frusemide. 4. Frusemide increased faecal potassium during the 48 h following administration and faecal water in the 24/48 h period. It also produced a hypopotassaemia. 5. Hydrochlorthiazide increased faecal chloride during the 24 h after administration. 6. Frusemide increased the intestinal transit time of both liquid (polyethylene glycol) and particulate (Cr2O3) markers.
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