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Author Horner, V.; Whiten, A.; Flynn, E.; de Waal, F.B.M.
Title Faithful replication of foraging techniques along cultural transmission chains by chimpanzees and children Type Journal Article
Year 2006 Publication Proceedings of the National Academy of Sciences of the United States of America Abbreviated Journal Proc. Natl. Acad. Sci. U.S.A.
Volume 103 Issue 37 Pages (down) 13878-13883
Keywords Animals; Child, Preschool; Humans; *Imitative Behavior; Pan troglodytes/*psychology
Abstract Observational studies of wild chimpanzees (Pan troglodytes) have revealed population-specific differences in behavior, thought to represent cultural variation. Field studies have also reported behaviors indicative of cultural learning, such as close observation of adult skills by infants, and the use of similar foraging techniques within a population over many generations. Although experimental studies have shown that chimpanzees are able to learn complex behaviors by observation, it is unclear how closely these studies simulate the learning environment found in the wild. In the present study we have used a diffusion chain paradigm, whereby a behavior is passed from one individual to the next in a linear sequence in an attempt to simulate intergenerational transmission of a foraging skill. Using a powerful three-group, two-action methodology, we found that alternative methods used to obtain food from a foraging device (“lift door” versus “slide door”) were accurately transmitted along two chains of six and five chimpanzees, respectively, such that the last chimpanzee in the chain used the same method as the original trained model. The fidelity of transmission within each chain is remarkable given that several individuals in the no-model control group were able to discover either method by individual exploration. A comparative study with human children revealed similar results. This study is the first to experimentally demonstrate the linear transmission of alternative foraging techniques by non-human primates. Our results show that chimpanzees have a capacity to sustain local traditions across multiple simulated generations.
Address Centre for Social Learning and Cognitive Evolution, School of Psychology, University of St. Andrews, Fife KY16 9JP, United Kingdom
Corporate Author Thesis
Publisher Place of Publication Editor
Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0027-8424 ISBN Medium
Area Expedition Conference
Notes PMID:16938863 Approved no
Call Number refbase @ user @ Serial 159
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Author Lee, R.D.
Title Rethinking the evolutionary theory of aging: transfers, not births, shape senescence in social species Type Journal Article
Year 2003 Publication Proceedings of the National Academy of Sciences of the United States of America Abbreviated Journal Proc Natl Acad Sci U S A
Volume 100 Issue 16 Pages (down) 9637-9642
Keywords Adaptation, Physiological; *Aging; Animals; *Biological Evolution; Demography; Economics; Environment; Fertility; Humans; Life Expectancy; Longevity; Models, Theoretical; Parturition; Population Dynamics; Population Growth; Reproduction
Abstract The classic evolutionary theory of aging explains why mortality rises with age: as individuals grow older, less lifetime fertility remains, so continued survival contributes less to reproductive fitness. However, successful reproduction often involves intergenerational transfers as well as fertility. In the formal theory offered here, age-specific selective pressure on mortality depends on a weighted average of remaining fertility (the classic effect) and remaining intergenerational transfers to be made to others. For species at the optimal quantity-investment tradeoff for offspring, only the transfer effect shapes mortality, explaining postreproductive survival and why juvenile mortality declines with age. It also explains the evolution of lower fertility, longer life, and increased investments in offspring.
Address Department of Demography, University of California, 2232 Piedmont Avenue, Berkeley, CA 94720-2120, USA. rlee@demog.berkeley.edu
Corporate Author Thesis
Publisher Place of Publication Editor
Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0027-8424 ISBN Medium
Area Expedition Conference
Notes PMID:12878733 Approved no
Call Number Equine Behaviour @ team @ Serial 5465
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Author Chase, I.D.; Tovey, C.; Spangler-Martin, D.; Manfredonia, M.
Title Individual differences versus social dynamics in the formation of animal dominance hierarchies Type Journal Article
Year 2002 Publication Proceedings of the National Academy of Sciences of the United States of America Abbreviated Journal Proc. Natl. Acad. Sci. U.S.A.
Volume 99 Issue 8 Pages (down) 5744-5749
Keywords Animals; *Behavior, Animal; Fishes; Humans; *Social Behavior; *Social Dominance
Abstract Linear hierarchies, the classical pecking-order structures, are formed readily in both nature and the laboratory in a great range of species including humans. However, the probability of getting linear structures by chance alone is quite low. In this paper we investigate the two hypotheses that are proposed most often to explain linear hierarchies: they are predetermined by differences in the attributes of animals, or they are produced by the dynamics of social interaction, i.e., they are self-organizing. We evaluate these hypotheses using cichlid fish as model animals, and although differences in attributes play a significant part, we find that social interaction is necessary for high proportions of groups with linear hierarchies. Our results suggest that dominance hierarchy formation is a much richer and more complex phenomenon than previously thought, and we explore the implications of these results for evolutionary biology, the social sciences, and the use of animal models in understanding human social organization.
Address Department of Sociology, State University of New York, Stony Brook, NY 11794-4356, USA. Ichase@notes.cc.sunysb.edu
Corporate Author Thesis
Publisher Place of Publication Editor
Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0027-8424 ISBN Medium
Area Expedition Conference
Notes PMID:11960030 Approved no
Call Number refbase @ user @ Serial 442
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Author Griffin, D.R.
Title Animals know more than we used to think Type
Year 2001 Publication Proceedings of the National Academy of Sciences of the United States of America Abbreviated Journal Proc. Natl. Acad. Sci. U.S.A.
Volume 98 Issue 9 Pages (down) 4833-4834
Keywords Animal Communication; Animals; Attention/physiology; Brain/physiology; Choice Behavior/physiology; Cognition/*physiology; Humans; Macaca mulatta/physiology/*psychology; Memory/*physiology; Optic Disk/physiology; Psychological Tests
Abstract
Address
Corporate Author Thesis
Publisher Place of Publication Editor
Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0027-8424 ISBN Medium
Area Expedition Conference
Notes PMID:11320232 Approved no
Call Number Equine Behaviour @ team @ Serial 2823
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Author Cheung, C.; Akiyama, T.E.; Ward, J.M.; Nicol, C.J.; Feigenbaum, L.; Vinson, C.; Gonzalez, F.J.
Title Diminished hepatocellular proliferation in mice humanized for the nuclear receptor peroxisome proliferator-activated receptor alpha Type Journal Article
Year 2004 Publication Cancer research Abbreviated Journal Cancer Res
Volume 64 Issue 11 Pages (down) 3849-3854
Keywords Animals; Anticholesteremic Agents/pharmacology; Carcinogens/pharmacology; Cell Division; DNA Replication/drug effects; Fatty Acids/metabolism; Hepatocytes/cytology/drug effects/metabolism/*physiology; Humans; Mice; Mice, Transgenic; Oxidation-Reduction; Peroxisome Proliferators/pharmacology; Pyrimidines/pharmacology; Receptors, Cytoplasmic and Nuclear/genetics/*physiology; Species Specificity; Transcription Factors/genetics/*physiology
Abstract Lipid-lowering fibrate drugs function as agonists for the nuclear receptor peroxisome proliferator-activated receptor alpha (PPARalpha). Sustained activation of PPARalpha leads to the development of liver tumors in rats and mice. However, humans appear to be resistant to the induction of peroxisome proliferation and the development of liver cancer by fibrate drugs. The molecular basis of this species difference is not known. To examine the mechanism determining species differences in peroxisome proliferator response between mice and humans, a PPARalpha-humanized mouse line was generated in which the human PPARalpha was expressed in liver under control of the tetracycline responsive regulatory system. The PPARalpha-humanized and wild-type mice responded to treatment with the potent PPARalpha ligand Wy-14643 as revealed by induction of genes encoding peroxisomal and mitochondrial fatty acid metabolizing enzymes and resultant decrease of serum triglycerides. However, surprisingly, only the wild-type mice and not the PPARalpha-humanized mice exhibited hepatocellular proliferation as revealed by elevation of cell cycle control genes, increased incorporation of 5-bromo-2'-deoxyuridine into hepatocyte nuclei, and hepatomegaly. These studies establish that following ligand activation, the PPARalpha-mediated pathways controlling lipid metabolism are independent from those controlling the cell proliferation pathways. These findings also suggest that structural differences between human and mouse PPARalpha are responsible for the differential susceptibility to the development of hepatocarcinomas observed after treatment with fibrates. The PPARalpha-humanized mice should serve as models for use in drug development and human risk assessment and to determine the mechanism of hepatocarcinogenesis of peroxisome proliferators.
Address Laboratory of Metabolism, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland 20892, USA
Corporate Author Thesis
Publisher Place of Publication Editor
Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0008-5472 ISBN Medium
Area Expedition Conference
Notes PMID:15172993 Approved no
Call Number refbase @ user @ Serial 74
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Author Kida, H.
Title [Ecology of influenza viruses in animals and the mechanism of emergence of new pandemic strains] Type Journal Article
Year 1997 Publication Nippon Rinsho. Japanese Journal of Clinical Medicine Abbreviated Journal Nippon Rinsho
Volume 55 Issue 10 Pages (down) 2521-2526
Keywords Animals; Bird Diseases/transmission; Birds; Horse Diseases/transmission; Horses; Humans; Influenza, Human/transmission/*veterinary; Swine; Swine Diseases/transmission; Zoonoses
Abstract Ecological studies on influenza viruses revealed that the hemagglutinin genes are introduced into new pandemic strains from viruses circulating in migratory ducks through domestic ducks and pigs in southern China. Experimental infection of pigs with 38 avian influenza virus strains with H1-H13 hemagglutinins showed that at least one strain of each HA subtype replicated in the upper respiratory tract of pigs. Co-infection of pigs with a swine virus and with an avian virus generated reassortant viruses. The results indicate that avian viruses of any subtype can contribute genes in the generation of reassortants. Virological surveillance revealed that influenza viruses in waterfowl reservoir are perpetuated year-by-year in the frozen lake water while ducks are absent.
Address Department of Disease Control, Hokkaido University Graduate School of Veterinary Medicine
Corporate Author Thesis
Publisher Place of Publication Editor
Language Japanese Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0047-1852 ISBN Medium
Area Expedition Conference
Notes PMID:9360367 Approved no
Call Number Equine Behaviour @ team @ Serial 2654
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Author Bentley, T.; Macky, K.; Edwards, J.
Title Injuries to New Zealanders participating in adventure tourism and adventure sports: an analysis of Accident Compensation Corporation (ACC) claims Type Journal Article
Year 2006 Publication The New Zealand Medical Journal Abbreviated Journal N Z Med J
Volume 119 Issue 1247 Pages (down) U2359
Keywords Adolescent; Adult; Aged; Athletic Injuries/*economics/*epidemiology; Back Injuries/epidemiology; Bicycling/economics/injuries; Female; Humans; Insurance Claim Reporting/*statistics & numerical data; Insurance, Liability/*utilization; Male; Middle Aged; Mountaineering/economics/injuries; New Zealand/epidemiology; *Risk-Taking; Skiing/economics/injuries; Sprains and Strains/epidemiology
Abstract AIMS: The aim of this study was to examine the involvement of adventure tourism and adventure sports activity in injury claims made to the Accident Compensation Corporation (ACC). METHODS: Epidemiological analysis of ACC claims for the period, July 2004 to June 2005, where adventure activities were involved in the injury. RESULTS: 18,697 adventure tourism and adventure sports injury claims were identified from the data, representing 28 activity sectors. Injuries were most common during the summer months, and were most frequently located in the major population centres. The majority of injuries were incurred by claimants in the 20-50 years age groups, although claimants over 50 years of age had highest claims costs. Males incurred 60% of all claims. Four activities (horse riding, mountain biking, tramping/hiking, and surfing) were responsible for approximately 60% of all adventure tourism and adventure sports-related injuries. Slips, trips, and falls were the most common injury initiating events, and injuries were most often to the back/spine, shoulder, and knee. CONCLUSIONS: These findings suggest the need to investigate whether regulatory intervention in the form of codes of practice for high injury count activities such as horse riding and mountain biking may be necessary. Health promotion messages and education programs should focus on these and other high-injury risk areas. Improved risk management practices are required for commercial adventure tourism and adventure sports operators in New Zealand if safety is to be improved across this sector.
Address Department of Management and International Business Massey University, Auckland. T.A.Bentley@massey.ac.nz
Corporate Author Thesis
Publisher Place of Publication Editor
Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 1175-8716 ISBN Medium
Area Expedition Conference
Notes PMID:17195852 Approved no
Call Number Serial 1843
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Author Pennisi, E.
Title Are out primate cousins 'conscious'? Type
Year 1999 Publication Science (New York, N.Y.) Abbreviated Journal Science
Volume 284 Issue 5423 Pages (down) 2073-2076
Keywords Animals; *Behavior, Animal; Cebus; *Consciousness; Empathy; Humans; Instinct; Intelligence; Learning; *Mental Processes; Pan troglodytes; *Primates
Abstract
Address
Corporate Author Thesis
Publisher Place of Publication Editor
Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0036-8075 ISBN Medium
Area Expedition Conference
Notes PMID:10409060 Approved no
Call Number Equine Behaviour @ team @ Serial 2843
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Author Selby, L.A.; Marienfeld, C.J.; Pierce, J.O.
Title The effects of trace elements on human and animal health Type Journal Article
Year 1970 Publication Journal of the American Veterinary Medical Association Abbreviated Journal J Am Vet Med Assoc
Volume 157 Issue 11 Pages (down) 1800-1808
Keywords Anemia, Hypochromic/veterinary; Animal Nutrition Physiology; Animals; Artiodactyla/*physiology; Chemistry; Cobalt/analysis/metabolism; Copper/analysis/metabolism; Deficiency Diseases/veterinary; Dogs/*physiology; Ecology; Horses/*physiology; Humans; Iodine/analysis/metabolism; Iron/analysis/metabolism; Manganese/analysis/metabolism; Nutritional Requirements; Selenium/metabolism; Trace Elements/*metabolism; Zinc/analysis/metabolism
Abstract
Address
Corporate Author Thesis
Publisher Place of Publication Editor
Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0003-1488 ISBN Medium
Area Expedition Conference
Notes PMID:4922190 Approved no
Call Number Equine Behaviour @ team @ Serial 2733
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Author Nicol, C.J.; Yoon, M.; Ward, J.M.; Yamashita, M.; Fukamachi, K.; Peters, J.M.; Gonzalez, F.J.
Title PPARgamma influences susceptibility to DMBA-induced mammary, ovarian and skin carcinogenesis Type Journal Article
Year 2004 Publication Carcinogenesis Abbreviated Journal Carcinogenesis
Volume 25 Issue 9 Pages (down) 1747-1755
Keywords 9,10-Dimethyl-1,2-benzanthracene/*toxicity; Animals; DNA Primers/chemistry; Disease Susceptibility; Female; Heterozygote; Humans; Mammary Neoplasms, Experimental/chemically induced/*pathology; Mice; Ovarian Neoplasms/chemically induced/*pathology; RNA, Messenger/genetics/metabolism; Receptors, Cytoplasmic and Nuclear/genetics/*physiology; Reverse Transcriptase Polymerase Chain Reaction; Skin Neoplasms/chemically induced/*pathology; Survival Rate; Transcription Factors/genetics/*physiology; Zinc Fingers
Abstract Peroxisome proliferator-activated receptor gamma (PPARgamma), a member of the nuclear receptor superfamily, plays a role in adipocyte differentiation, type II diabetes, macrophage response to inflammation and is suggested to influence carcinogen-induced colon cancer. Studies done in vitro and in vivo also revealed that PPARgamma ligands might promote differentiation and/or regression of mammary tumors. To directly evaluate the role of PPARgamma in mammary carcinogenesis, PPARgamma wild-type (+/+) or heterozygous (+/-) mice were administered 1 mg 7,12-dimethylbenz[a]anthracene (DMBA) by gavage once a week for 6 weeks and followed for a total of 25 weeks. Compared with congenic PPARgamma(+/+) littermate controls, PPARgamma(+/-) mice had early evidence for increased susceptibility to DMBA-mediated carcinogenesis based on a 1.6-fold increase in the percentage of mice with skin papillomas, as well as a 1.7-fold increase in the numbers of skin papillomas per mouse (P < 0.05). Similarly, PPARgamma(+/-) mice also had a 1.5-fold decreased survival rate (P = 0.059), and a 1.7-fold increased incidence of total tumors per mouse (P < 0.01). Moreover, PPARgamma(+/-) mice had an almost 3-fold increase in mammary adenocarcinomas (P < 0.05), an over 3-fold increase in ovarian granulosa cell carcinomas (P < 0.05), an over 3-fold increase in malignant tumors (P < 0.02) and a 4.6-fold increase in metastatic incidence. These results are the first to demonstrate an increased susceptibility in vivo of PPARgamma haploinsufficiency to DMBA-mediated carcinogenesis and suggest that PPARgamma may act as a tumor modifier of skin, ovarian and breast cancers. The data also support evidence suggesting a beneficial role for PPARgamma-specific ligands in the chemoprevention of mammary, ovarian and skin carcinogenesis.
Address Laboratory of Metabolism, Center for Cancer Research, National Cancer Institute, Bethesda, MD 20892, USA
Corporate Author Thesis
Publisher Place of Publication Editor
Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0143-3334 ISBN Medium
Area Expedition Conference
Notes PMID:15073042 Approved no
Call Number refbase @ user @ Serial 76
Permanent link to this record