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Author Boray, J.C.
Title Experimental fascioliasis in Australia Type Journal Article
Year 1969 Publication (up) Advances in Parasitology Abbreviated Journal Adv Parasitol
Volume 7 Issue Pages 95-210
Keywords Adaptation, Biological; Adaptation, Physiological; Animal Nutrition Physiology; Animals; Animals, Laboratory; Australia; Cattle; *Cattle Diseases/pathology; Climate; *Disease Vectors; Ecology; Electron Transport; Estivation; Fasciola hepatica/enzymology/*growth & development/metabolism/physiology; Fascioliasis/epidemiology/immunology/*prevention & control/veterinary; Glycolysis; Guinea Pigs; Horses; Humans; Larva/growth & development/physiology; Marsupialia; Metamorphosis, Biological; Mice; New Guinea; New Zealand; Parasite Egg Count; Rats; Seasons; Sheep; *Sheep Diseases/pathology
Abstract
Address
Corporate Author Thesis
Publisher Place of Publication Editor
Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0065-308X ISBN Medium
Area Expedition Conference
Notes PMID:4935272 Approved no
Call Number Equine Behaviour @ team @ Serial 2744
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Author Komar, N.
Title West Nile virus: epidemiology and ecology in North America Type Journal Article
Year 2003 Publication (up) Advances in Virus Research Abbreviated Journal Adv Virus Res
Volume 61 Issue Pages 185-234
Keywords Animals; Bird Diseases/virology; Birds/virology; Culex/virology; Disease Reservoirs; Ecosystem; Epidemiology, Molecular; Horse Diseases/virology; Horses/virology; Humans; Insect Vectors; North America/epidemiology; Risk Factors; West Nile Fever/*epidemiology/transmission/veterinary; West Nile virus/genetics
Abstract
Address Centers for Disease Control and Prevention, Division of Vector-Borne Infectious Diseases, Fort Collins, Colorado 80522, USA
Corporate Author Thesis
Publisher Place of Publication Editor
Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0065-3527 ISBN Medium
Area Expedition Conference
Notes PMID:14714433 Approved no
Call Number Equine Behaviour @ team @ Serial 2638
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Author Scherer, W.F.; Madalengoitia, J.; Flores, W.; Acosta, M.
Title Ecologic studies of Venezuelan encephalitis virus in Peru during 1970-1971 Type Journal Article
Year 1975 Publication (up) American Journal of Epidemiology Abbreviated Journal Am J Epidemiol
Volume 101 Issue 4 Pages 347-355
Keywords Animals; Antibodies, Viral; Cricetinae/immunology; Culicidae/microbiology; *Disease Vectors; Ecology; *Encephalitis Virus, Venezuelan Equine/immunology/isolation & purification; Encephalomyelitis, Equine/immunology/microbiology/transmission; Female; Hemagglutination Inhibition Tests; Horses/immunology; Humans; Neutralization Tests; Peru
Abstract Venezuelan encephalitis (VE) virus has intermittently produced epidemics and equine epizootics on the dry Pacific coastal plain of Peru since at least the 1930's. However, evidence that the virus exists in the Amazon region of Peru to the east of the Andes mountains was not obtained until antibodies were found in human sera collected in 1965, and 10 strains of the virus were isolated in a forest near the city of Iquitos, Peru during February and March 1971. Eight strains came from mosquitoes and two from dead sentinel hamsters. Three hamsters exposed in forests near Iquitos developed VE virus antibodies suggesting that hamster-benign strains also exist there. Antibody tests of equine sera revealed no evidence that VE virus was actively cycling during the late 1950's or 1960's in southern coastal Peru, where equine epizootics had occurred in the 1930's and 1940's. In northern coastal Peru bordering Ecuador, antibodies were present in equine sera, presumably residual from the 1969 outbreak caused by subtype I virus, since neutralizing antibody titers were higher to subtype I virus than to subtypes III or IV. No VE virus was detected in this northern region during the dry season of 1970 by use of sentinel hamsters. The possibility is considered that VE epidemics and equine epizootics on the Pacific coast of Peru are caused by movements of virus in infected vertebrates traversing Andean passes or in infected vertebrates or mosquitoes carried in airplanes from the Amazon region.
Address
Corporate Author Thesis
Publisher Place of Publication Editor
Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0002-9262 ISBN Medium
Area Expedition Conference
Notes PMID:235838 Approved no
Call Number Equine Behaviour @ team @ Serial 2705
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Author Sudia, W.D.; Fernandez, L.; Newhouse, V.F.; Sanz, R.; Calisher, C.H.
Title Arbovirus vector ecology studies in Mexico during the 1972 Venezuelan equine encephalitis outbreak Type Journal Article
Year 1975 Publication (up) American Journal of Epidemiology Abbreviated Journal Am J Epidemiol
Volume 101 Issue 1 Pages 51-58
Keywords Animals; Arboviruses/isolation & purification; Culicidae/microbiology; Disease Vectors/*microbiology; Ecology; Encephalitis Virus, St. Louis/isolation & purification; Encephalitis Virus, Venezuelan Equine/*isolation & purification; Encephalitis Virus, Western Equine/isolation & purification; Encephalomyelitis, Equine/epidemiology/*transmission/veterinary; Horse Diseases/epidemiology/*transmission; Horses; Insect Vectors/microbiology; Mexico
Abstract Virus vector studies were conducted in the States of Durango, Chihuahua, and Tamaulipas, Mexico, in June and July 1972. Apparently only a low level of Venzuelan equine encephalitis (VEE) virus transmission to equines occured at the time of the study, and the infection was restricted to areas which had not experienced overt activity during the preceding year. The low level of infection was associated with a scarcity of mosquitoes. The IB (epidemic) strain of VEE virus was isolated from two pools of Anopheles pseudopunctipennis (Theo.) and the blood of one symptomatic equine. The low mosquito population, the relatively few equine cases observed, and the absence of reports of VEE human disease from the outbreak area suggested VEE virus persistence through a low-level mosquito-equine transmission cycle. Other studies have already indicated that wild vertebrates play no more than a minor role in outbreaks of epidemic VEE. Mosquito collections made in areas of the states of Durango, Chihuahua, and Tamaulipas, where considerable epidemic activity of VEE had occurred in 1971, failed to reveal evidence of VEE virus persistence. Twenty-nine ioslations of other arboviruses were also made in these studies: including 22 of St. Louis encephalitis virus (SLE), 2 of Flanders virus, 1 of Turlock virus, 1 of Trivittatus virus of the California Group, 1 of western equine encephalitis virus (VEE), and 2 (from Santa Rose) which possibly represent a hitherto unknown virus in the Bunyamwera Group. These are the first reports of SLE virus isolations from mosquitoes in Mexico, and the first demonstration of Trivittatus, VEE Turlock and Flanders viruses in Mexico from any source.
Address
Corporate Author Thesis
Publisher Place of Publication Editor
Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0002-9262 ISBN Medium
Area Expedition Conference
Notes PMID:235213 Approved no
Call Number Equine Behaviour @ team @ Serial 2706
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Author Menges, R.W.; Furcolow, M.L.; Selby, L.A.; Habermann, R.T.; Smith, C.D.
Title Ecologic studies of histoplasmosis Type Journal Article
Year 1967 Publication (up) American Journal of Epidemiology Abbreviated Journal Am J Epidemiol
Volume 85 Issue 1 Pages 108-119
Keywords Adolescent; Adult; Animals; Antibodies/*analysis; Carnivora; Cats; Cattle; Child; Child, Preschool; Dogs; Ecology; Female; Fluorescent Antibody Technique; Histoplasma/isolation & purification; Histoplasmin; Histoplasmosis/*epidemiology/*immunology; Horses; Humans; Infant; Infant, Newborn; Kansas; Male; Marsupialia; Mice; Middle Aged; Missouri; Rabbits; Skin Tests; *Soil Microbiology; Swine
Abstract
Address
Corporate Author Thesis
Publisher Place of Publication Editor
Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0002-9262 ISBN Medium
Area Expedition Conference
Notes PMID:5334640 Approved no
Call Number Equine Behaviour @ team @ Serial 2747
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Author Nicol, C.J.; Adachi, M.; Akiyama, T.E.; Gonzalez, F.J.
Title PPARgamma in endothelial cells influences high fat diet-induced hypertension Type Journal Article
Year 2005 Publication (up) American journal of hypertension : journal of the American Society of Hypertension Abbreviated Journal Am J Hypertens
Volume 18 Issue 4 Pt 1 Pages 549-556
Keywords Administration, Oral; Animals; Antihypertensive Agents/pharmacology; Blood Pressure/drug effects; Diabetes Mellitus, Type 2/physiopathology; Dietary Fats/*administration & dosage/pharmacology; Dose-Response Relationship, Drug; Endothelial Cells/*metabolism; Female; Heart Rate/drug effects; Hypertension/*etiology; Ligands; Male; Mice; Mice, Knockout; PPAR gamma/*metabolism; Sodium Chloride/administration & dosage/pharmacology; Thiazolidinediones/pharmacology
Abstract BACKGROUND: Peroxisome proliferator-activated receptor gamma (PPARgamma) ligands improve human hypertension. However, the mechanism and site of this effect remains unknown, confounded by PPARgamma expression in many cell types, including endothelial cells (ECs). METHODS: To evaluate the vascular role of PPARgamma we used a conditional null mouse model. Specific disruption of PPARgamma in ECs was created by crossing Tie2-Cre+ transgenic (T2T+) and PPARgamma-floxed (fl/fl) mice to generate PPARgamma (fl/fl)T2T+ (PPARgamma E-null) mice. Conscious 8- to 12-week-old congenic PPARgamma (fl/fl)Cre- (wild type) and PPARgamma E-null mice were examined for changes in systolic blood pressure (BP) and heart rate (HR), untreated, after 2 months of salt-loading (drinking water), and after treatment for 3 months with high fat (HF) diet alone or supplemented during the last 2 weeks with rosiglitazone (3 mg/kg/d). RESULTS: Untreated PPARgamma E-nulls were phenotypically indistinguishable from wild-type littermates. However, compared to similarly treated wild types, HF-treated PPARgamma E-nulls had significantly elevated systolic BP not seen after normal diet or salt-loading. Despite sex-dependent baseline differences, salt-loaded and HF-treated PPARgamma E-nulls of either sex had significantly elevated HR versus wild types. Interestingly, rosiglitazone improved serum insulin levels, but not HF diet-induced hypertension, in PPARgamma E-null mice. CONCLUSIONS: These results suggest that PPARgamma in ECs not only is an important regulator of hypertension and HR under stressed conditions mimicking those arising in type 2 diabetics, but also mediates the antihypertensive effects of rosiglitazone. These data add evidence supporting a beneficial role for PPARgamma-specific ligands in the treatment of hypertension, and suggest therapeutic strategies targeting ECs may prove useful.
Address Laboratory of Metabolism, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland 20892, USA
Corporate Author Thesis
Publisher Place of Publication Editor
Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0895-7061 ISBN Medium
Area Expedition Conference
Notes PMID:15831367 Approved no
Call Number refbase @ user @ Serial 69
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Author de Waal, F.B.; Uno, H.; Luttrell, L.M.; Meisner, L.F.; Jeannotte, L.A.
Title Behavioral retardation in a macaque with autosomal trisomy and aging mother Type Journal Article
Year 1996 Publication (up) American journal of mental retardation : AJMR Abbreviated Journal Am J Ment Retard
Volume 100 Issue 4 Pages 378-390
Keywords Animals; *Behavior, Animal; Brain/physiopathology; Female; Hydrocephalus/complications; Longitudinal Studies; Macaca mulatta/*genetics; Magnetic Resonance Imaging; Male; *Maternal Age; Psychomotor Disorders/*etiology; Social Behavior; Trisomy/*genetics; X Chromosome
Abstract The social development of a female rhesus monkey (Macaca mulatta) was followed from the day of birth until her death, at age 32 months. The subject, born to an older mother, had an extra autosome (karyotype: 43, XX, +18), an affliction that came about spontaneously. MRI scans revealed that she was also hydrocephalic. Compared to 23 female monkeys growing up under identical conditions, the subject showed serious motor deficiencies, a dramatic delay in the development of social behavior, poorly established dominance relationships, and greater than usual dependency on mother and kin. The subject was well-integrated into the social group, however.
Address University of Wisconsin-Madison, USA
Corporate Author Thesis
Publisher Place of Publication Editor
Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0895-8017 ISBN Medium
Area Expedition Conference
Notes PMID:8718992 Approved no
Call Number refbase @ user @ Serial 205
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Author Palagi, E.; Cordoni, G.; Borgognini Tarli, S.
Title Possible roles of consolation in captive chimpanzees (Pan troglodytes) Type Journal Article
Year 2006 Publication (up) American Journal of Physical Anthropology Abbreviated Journal Am J Phys Anthropol
Volume 129 Issue 1 Pages 105-111
Keywords Animals; Animals, Zoo/*physiology; Empathy; Female; Male; Pan troglodytes/*physiology; *Social Behavior; Stress/physiopathology/veterinary
Abstract Empathy is a necessary prerequisite for the occurrence of consolation. The term “consolation” contains a hypothesis about function, which is distress alleviation. The present study aims to confirm the occurrence of consolation in captive chimpanzees via the post-conflict/matched-control method (PC-MC) and to suggest its possible roles. We collected 273 PC-MC pairs in the group of Pan troglodytes housed in the ZooParc de Beauval (France). We confirmed the presence of consolatory contacts (mean level of consolation, 49.5% +/- 22.3% SEM) in the colony. Consolation rates were significantly higher than reconciliation levels (mean level of reconciliation, 28.9% +/- 16.8% SEM). The level of consolation was greater in the absence of reconciliation than in the presence of it, suggesting that consolation might be an alternative behavior. As friendship and relatedness did not influence the occurrence of consolation, they did not seem to be the best prerequisites for this behavioral mechanism, at least in this chimpanzee colony. Affinitive contacts with third parties were significantly more frequent when the victim called attention to itself during severe aggressions by screaming. These high-pitched sounds seem to be useful in eliciting aid from conspecifics, as occurs in young humans. The occurrence of consolation reduced the likelihood of further attacks among group-members. From this perspective, both victims and consolers most likely gain potential advantages by interacting with each other when aggression is particularly severe, reconciliation is not immediate, and consequently social stress reaches high levels.
Address Centro Interdipartimentale Museo di Storia Naturale e del Territorio, Universita di Pisa, 56010 Calci, Italy. betta.palagi@museo.unipi.it
Corporate Author Thesis
Publisher Place of Publication Editor
Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0002-9483 ISBN Medium
Area Expedition Conference
Notes PMID:16229027 Approved no
Call Number refbase @ user @ Serial 2871
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Author Previc, F.H.
Title Thyroid hormone production in chimpanzees and humans: implications for the origins of human intelligence Type Journal Article
Year 2002 Publication (up) American Journal of Physical Anthropology Abbreviated Journal Am J Phys Anthropol
Volume 118 Issue 4 Pages 402-3; discussion 404-5
Keywords Animals; Humans; *Intelligence; Pan troglodytes/*metabolism; Species Specificity; Thyroid Hormones/*biosynthesis
Abstract
Address Northrop Grumman Information Technology, San Antonio, Texas 78228, USA. fred.previc@brooks.af.mil
Corporate Author Thesis
Publisher Place of Publication Editor
Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0002-9483 ISBN Medium
Area Expedition Conference
Notes PMID:12124921 Approved no
Call Number Equine Behaviour @ team @ Serial 4108
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Author Brosnan, S.F.; Freeman, C.; De Waal, F.B.M.
Title Partner's behavior, not reward distribution, determines success in an unequal cooperative task in capuchin monkeys Type Journal Article
Year 2006 Publication (up) American journal of primatology Abbreviated Journal Am. J. Primatol.
Volume 68 Issue 7 Pages 713-724
Keywords Animals; Behavior, Animal/*physiology; Cebus/*physiology; *Cooperative Behavior; Female; Food Preferences/physiology; Male; *Reward
Abstract It was recently demonstrated that capuchin monkeys notice and respond to distributional inequity, a trait that has been proposed to support the evolution of cooperation in the human species. However, it is unknown how capuchins react to inequitable rewards in an unrestricted cooperative paradigm in which they may freely choose both whether to participate and, within the bounds of their partner's behavior, which reward they will receive for their participation. We tested capuchin monkeys with such a design, using a cooperative barpull, which has been used with great success in the past. Contrary to our expectations, the equity of the reward distribution did not affect success or pulling behavior. However, the behavior of the partner in an unequal situation did affect overall success rates: pairs that had a tendency to alternate which individual received the higher-value food in unequal reward situations were more than twice as successful in obtaining rewards than pairs in which one individual dominated the higher-value food. This ability to equitably distribute rewards in inherently biased cooperative situations has profound implications for activities such as group hunts, in which multiple individuals work together for a single, monopolizable reward.
Address Living Links Center, Yerkes National Primate Research Center, Emory University, Atlanta, Georgia, USA. sbrosna@emory.edu
Corporate Author Thesis
Publisher Place of Publication Editor
Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0275-2565 ISBN Medium
Area Expedition Conference
Notes PMID:16786518 Approved no
Call Number refbase @ user @ Serial 160
Permanent link to this record