Records |
Author |
Connor, R.C.; Mann, J.; Tyack, P.L.; Whitehead, H. |
Title |
Social evolution in toothed whales |
Type |
Journal Article |
Year |
1998 |
Publication |
Trends in Ecology & Evolution |
Abbreviated Journal |
Trends. Ecol. Evol |
Volume |
13 |
Issue |
6 |
Pages |
228-232 |
Keywords |
odontocetes; toothed whales; social evolution; communication; bottlenose dolphins; sperm whales; long-term studies; foraging |
Abstract |
Two contrasting results emerge from comparisons of the social systems of several odontocetes with terrestrial mammals. Researchers have identified remarkable convergence in prominent features of the social systems of odontocetes such as the sperm whale and bottlenose dolphin with a few well-known terrestrial mammals such as the elephant and chimpanzee. In contrast, studies on killer whales and Baird's beaked whale reveal novel social solutions to aquatic living. The combination of convergent and novel features in odontocete social systems promise a more general understanding of the ecological determinants of social systems in both terrestrial and aquatic habitats, as well as the relationship between relative brain size and social evolution. |
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0169-5347 |
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Equine Behaviour @ team @ |
Serial |
4789 |
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Author |
Connor, R.C. |
Title |
Altruism among non-relatives: alternatives to the 'Prisoner's Dilemma' |
Type |
Journal Article |
Year |
1995 |
Publication |
Trends in Ecology & Evolution |
Abbreviated Journal |
Trends Ecol Evol |
Volume |
10 |
Issue |
2 |
Pages |
84-86 |
Keywords |
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Abstract |
Triver's model of reciprocal altruism, and its descendants based on the Prisoner's Dilemma model, have dominated thinking about cooperation and altruism between non-relatives. However, there are three alternative models of altruism directed to non-relatives. These models, which are not based on the Prisoner's Dilemma, may explain a variety of phenomena, from allogrooming among impala to helping by non-relatives in cooperatively breeding birds and mammals. |
Address |
Division of Biological Sciences and The Michigan Society of Fellows, Museum of Zoology, University of Michigan, Ann Arbor MI, 48109, USA |
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0169-5347 |
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Notes |
PMID:21236964 |
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no |
Call Number |
Equine Behaviour @ team @ |
Serial |
5407 |
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Author |
Hampton, R.R.; Healy, S.D.; Shettleworth, S.J.; Kamil, A.C. |
Title |
Neuroecologists' are not made of straw |
Type |
Journal Article |
Year |
2002 |
Publication |
Trends in Cognitive Sciences |
Abbreviated Journal |
Trends. Cognit. Sci. |
Volume |
6 |
Issue |
1 |
Pages |
6-7 |
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Address |
Laboratory of Neuropsychology, NIH--NIMH, Building 49, Room 1B-80, 20892-4415, Bethesda, MD, USA |
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1364-6613 |
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PMID:11849608 |
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no |
Call Number |
refbase @ user @ |
Serial |
371 |
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Author |
Gomez, J.-C. |
Title |
Species comparative studies and cognitive development |
Type |
Journal Article |
Year |
2005 |
Publication |
Trends in Cognitive Sciences |
Abbreviated Journal |
Trends. Cognit. Sci. |
Volume |
9 |
Issue |
3 |
Pages |
118-125 |
Keywords |
Animals; Attention/physiology; Brain/*growth & development; Child, Preschool; Cognition/*physiology; Concept Formation/physiology; Dogs; Evolution; Fixation, Ocular; Gorilla gorilla; Humans; Infant; Learning/*physiology; Macaca mulatta; Mental Recall/physiology; Personal Construct Theory; Psychomotor Performance/physiology; Species Specificity |
Abstract |
The comparative study of infant development and animal cognition brings to cognitive science the promise of insights into the nature and origins of cognitive skills. In this article, I review a recent wave of comparative studies conducted with similar methodologies and similar theoretical frameworks on how two core components of human cognition--object permanence and gaze following--develop in different species. These comparative findings call for an integration of current competing accounts of developmental change. They further suggest that evolution has produced developmental devices capable at the same time of preserving core adaptive components, and opening themselves up to further adaptive change, not only in interaction with the external environment, but also in interaction with other co-developing cognitive systems. |
Address |
Scottish Primate Research Group, School of Psychology, University of St Andrews, St Andrews, Fife KY15 9JU, UK |
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ISSN |
1364-6613 |
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Notes |
PMID:15737820 |
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no |
Call Number |
Equine Behaviour @ team @ |
Serial |
2851 |
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Author |
Legare, C.H.; Nielsen, M. |
Title |
Imitation and Innovation: The Dual Engines of Cultural Learning |
Type |
Journal Article |
Year |
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Publication |
Trends in Cognitive Sciences |
Abbreviated Journal |
Trends in Cognitive Sciences |
Volume |
19 |
Issue |
11 |
Pages |
688-699 |
Keywords |
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Abstract |
Imitation and innovation work in tandem to support cultural learning in children and facilitate our capacity for cumulative culture. Here we propose an integrated theoretical account of how the unique demands of acquiring instrumental skills and cultural conventions provide insight into when children imitate, when they innovate, and to what degree. For instrumental learning, with an increase in experience, high fidelity imitation decreases and innovation increases. By contrast, for conventional learning, imitative fidelity stays high, regardless of experience, and innovation stays low. We synthesize cutting edge research on the development of imitative flexibility and innovation to provide insight into the social learning mechanisms underpinning the uniquely human mind. |
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Elsevier |
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1364-6613 |
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doi: 10.1016/j.tics.2015.08.005 |
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no |
Call Number |
Equine Behaviour @ team @ |
Serial |
5931 |
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Author |
Van Schaik, C.P.; Isler, K.; Burkart, J.M. |
Title |
Explaining brain size variation: from social to cultural brain |
Type |
Journal Article |
Year |
2012 |
Publication |
Trends Ecol Evol |
Abbreviated Journal |
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Volume |
16 |
Issue |
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Pages |
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Equine Behaviour @ team @ Van Schaik2012 |
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6304 |
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Author |
Wells, P.G.; Bhuller, Y.; Chen, C.S.; Jeng, W.; Kasapinovic, S.; Kennedy, J.C.; Kim, P.M.; Laposa, R.R.; McCallum, G.P.; Nicol, C.J.; Parman, T.; Wiley, M.J.; Wong, A.W. |
Title |
Molecular and biochemical mechanisms in teratogenesis involving reactive oxygen species |
Type |
Journal Article |
Year |
2005 |
Publication |
Toxicology and applied pharmacology |
Abbreviated Journal |
Toxicol Appl Pharmacol |
Volume |
207 |
Issue |
2 Suppl |
Pages |
354-366 |
Keywords |
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Abstract |
Developmental pathologies may result from endogenous or xenobiotic-enhanced formation of reactive oxygen species (ROS), which oxidatively damage cellular macromolecules and/or alter signal transduction. This minireview focuses upon several model drugs (phenytoin, thalidomide, methamphetamine), environmental chemicals (benzo[a]pyrene) and gamma irradiation to examine this hypothesis in vivo and in embryo culture using mouse, rat and rabbit models. Embryonic prostaglandin H synthases (PHSs) and lipoxygenases bioactivate xenobiotics to free radical intermediates that initiate ROS formation, resulting in oxidation of proteins, lipids and DNA. Oxidative DNA damage and embryopathies are reduced in PHS knockout mice, and in mice treated with PHS inhibitors, antioxidative enzymes, antioxidants and free radical trapping agents. Thalidomide causes embryonic DNA oxidation in susceptible (rabbit) but not resistant (mouse) species. Embryopathies are increased in mutant mice deficient in the antioxidative enzyme glucose-6-phosphate dehydrogenase (G6PD), or by glutathione (GSH) depletion, or inhibition of GSH peroxidase or GSH reductase. Inducible nitric oxide synthase knockout mice are partially protected. Inhibition of Ras or NF-kB pathways reduces embryopathies, implicating ROS-mediated signal transduction. Atm and p53 knockout mice deficient in DNA damage response/repair are more susceptible to xenobiotic or radiation embryopathies, suggesting a teratological role for DNA damage, consistent with enhanced susceptibility to methamphetamine in ogg1 knockout mice with deficient repair of oxidative DNA damage. Even endogenous embryonic oxidative stress carries a risk, since untreated G6PD- or ATM-deficient mice have increased embryopathies. Thus, embryonic processes regulating the balance of ROS formation, oxidative DNA damage and repair, and ROS-mediated signal transduction may be important determinants of teratological risk. |
Address |
Faculty of Pharmacy, University of Toronto, Toronto, Ontario, Canada; Department of Pharmacology, University of Toronto, Toronto, Ontario, Canada |
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English |
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Series Editor |
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Abbreviated Series Title |
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Edition |
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ISSN |
0041-008X |
ISBN |
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Notes |
PMID:16081118 |
Approved |
no |
Call Number |
refbase @ user @ |
Serial |
68 |
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Author |
Guo, G.L.; Moffit, J.S.; Nicol, C.J.; Ward, J.M.; Aleksunes, L.A.; Slitt, A.L.; Kliewer, S.A.; Manautou, J.E.; Gonzalez, F.J. |
Title |
Enhanced acetaminophen toxicity by activation of the pregnane X receptor |
Type |
Journal Article |
Year |
2004 |
Publication |
Toxicological sciences : an official journal of the Society of Toxicology |
Abbreviated Journal |
Toxicol Sci |
Volume |
82 |
Issue |
2 |
Pages |
374-380 |
Keywords |
Acetaminophen/pharmacokinetics/*toxicity; Analgesics, Non-Narcotic/pharmacokinetics/*toxicity; Animals; Aryl Hydrocarbon Hydroxylases/biosynthesis; Biotransformation; Blotting, Northern; Chromatography, High Pressure Liquid; Cytochrome P-450 CYP3A; Membrane Proteins; Mice; Mice, Knockout; Oxidoreductases, N-Demethylating/biosynthesis; Pregnenolone Carbonitrile/pharmacology; Receptors, Cytoplasmic and Nuclear/*drug effects; Receptors, Steroid/*drug effects; Sulfhydryl Compounds/metabolism |
Abstract |
The pregnane X receptor (PXR) is a ligand-activated transcription factor and member of the nuclear receptor superfamily. Activation of PXR represents an important mechanism for the induction of cytochrome P450 3A (CYP3A) enzymes that can convert acetaminophen (APAP) to its toxic intermediate metabolite, N-acetyl-p-benzoquinone imine (NAPQI). Therefore, it was hypothesized that activation of PXR plays a major role in APAP-induced hepatotoxicity. Pretreatment with the PXR activator, pregnenolone 16alpha-carbonitrile (PCN), markedly enhanced APAP-induced hepatic injury, as revealed by increased serum ALT levels and hepatic centrilobular necrosis, in wild-type but not in PXR-null mice. Further analysis showed that following PCN treatment, PXR-null mice had lower CYP3A11 expression, decreased NAPQI formation, and increased maintenance of hepatic glutathione content compared to wild-type mice. Thus, these results suggest that PXR plays a critical role in APAP-induced hepatic toxicity, probably by inducing CYP3A11 expression and hence increasing bioactivation. |
Address |
Laboratory of Metabolism, CCR, NCI, NIH, Bethesda, Maryland 20892, USA |
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English |
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1096-6080 |
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PMID:15456926 |
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no |
Call Number |
refbase @ user @ |
Serial |
71 |
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Author |
Sloet van Oldruitenborgh-Oosterbaan, M.M.; Blok, M.B.; Begeman, L.; Kamphuis, M.C.D.; Lameris, M.C.; Spierenburg, A.J.; Lashley, M.J.J.O. |
Title |
Workload and stress in horses: comparison in horses ridden deep and round ('rollkur') with a draw rein and horses ridden in a natural frame with only light rein contact |
Type |
Journal Article |
Year |
2006 |
Publication |
Tijdschrift Voor Diergeneeskunde |
Abbreviated Journal |
Tijdschr Diergeneeskd |
Volume |
131 |
Issue |
5 |
Pages |
152-157 |
Keywords |
Animal Husbandry/methods; Animals; Biomechanics; Blood Glucose/analysis; Female; Heart Rate/physiology; Hematocrit/veterinary; Horses/blood/*physiology; Hydrocortisone/blood; Lactic Acid/blood; Physical Conditioning, Animal/adverse effects/*physiology; Stress, Physiological/blood/etiology/veterinary |
Abstract |
'Rollkur' or 'overbending' is the low and deep riding of a dressage horse during training or warming up. Lately, this technique has been criticized, and not necessarily objectively, on welfare grounds. To be able to evaluate these criticisms, more needs to be known about the workload and stress of horses being ridden 'rollkur'. The aim of the present study was to compare the workload of eight riding-school horses when being ridden deep and round with a draw rein ('rollkur') and when being ridden in a natural frame with only light rein contact ('free'). Workload (as measured by heart rate and blood lactate concentration) was slightly higher when horses were ridden 'rollkur' than when they were ridden 'free'. There were no differences in packed cell volume, or glucose and cortisol concentrations. No signs of uneasiness or stress could be determined when the horses were ridden 'rollkur'. Subjectively, all horses improved their way of moving during 'rollkur' and were more responsive to their rider. |
Address |
Department of Equine Sciences, Faculty of Veterinary Medicine, Utrecht University, Yalelaan 12, 3584 CM Utrecht, The Netherlands. m.sloet@vet.uu.nl |
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0040-7453 |
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Notes |
PMID:16532786 |
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no |
Call Number |
Equine Behaviour @ team @ |
Serial |
5638 |
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Author |
Lachmann M.; Bergstrom C.T. |
Title |
Signalling among Relatives II. Beyond the Tower of Babel |
Type |
Journal Article |
Year |
1998 |
Publication |
Theoretical Population Biology |
Abbreviated Journal |
Theor. Pop. Biol. |
Volume |
54 |
Issue |
2 |
Pages |
146-160 |
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Abstract |
Models of costly signalling are commonly employed in evolutionary biology in order to explain how honest communication between individuals with conflicting interests can be stable. These models have focused primarily on a single type of honest signalling equilibrium, the separating equilibrium in which any two different signallers send distinct signals, thereby providing signal receivers with complete information. In this paper, we demonstrate that in signalling among relatives (modelled using the Sir Philip Sidney game), there is not one but a large number of possible signalling equilibria, most of which are pooling equilibria in which different types of signallers may share a common signal. We prove that in a general Sir Philip Sidney game, any partition of signallers into equi-signalling classes can have a stable signalling equilibrium if and only if it is a contiguous partition, and provide examples of such partitions. A similar (but slightly stricter) condition is shown to hold when signals are transmitted through a medium with signalling error. These results suggest a solution to a problem faced by previous signalling theory models: when we consider the separating equilibrium, signal cost is independent of the frequency of individuals sending that signal and, consequently, even very rare signaller types can drastically affect signal cost. Here, we show that by allowing these rare signallers to pool with more common signallers, signal cost can be greatly reduced. Copyright 1998 Academic Press. |
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refbase @ user @ |
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560 |
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