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Bouman Jg,. (1979). Does inbreeding ocur in free living horses? In L. E. M. de Boer, J. Bouman, & I. Bouman (Eds.), Genetics and Hereditary Diseases of the Przewalski horse (pp. 125–132). Rotterdam: Foundation for the Preservation and Protection of the Przewalski Horse.
Abstract: Boer, Leobert E.M. de, and Jan & Inge Bouman, eds. Genetics and Hereditary Diseases of the Przewalski Horse. Rotterdam: Foundation for the Preservation and Protection of the Przewalski Horse 1979. 176p, several photos and charts. Reproduced from typescript, as issued. Very good paperbound (covers shelf-soiled). ** Publishes 18 papers given at the Arnhem Study-Conference of 1978, on many aspects of breeding Przewalski horses, their behavior, etc.
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Bouman-Heinsdijk I,. (1979). Does inbreeding occur in free living horses? In L. E. M. de Boer, J. Bouman, & I. Bouman (Eds.), Genetics and Hereditary Diseases of the Przewalski Horse. Rotterdam: Foundation for the Preservation and Protection of the Przewalski Horse.
Abstract: Boer, Leobert E.M. de, and Jan & Inge Bouman, eds. Genetics and Hereditary Diseases of the Przewalski Horse. Rotterdam: Foundation for the Preservation and Protection of the Przewalski Horse 1979. 176p, several photos and charts. Reproduced from typescript, as issued. Very good paperbound (covers shelf-soiled). ** Publishes 18 papers given at the Arnhem Study-Conference of 1978, on many aspects of breeding Przewalski horses, their behavior, etc.
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Houpt, K. A., & Kusonose, R. (2000). Genetic of behaviour. In A. T. Bowling, & A. Ruvinsky (Eds.), Genetics of the Horse (pp. 281–306). Wallingford Oxfordshire: Cab Intl.
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Ricard, A., & Chanu, I. (2001). Genetic parameters of eventing horse competition in France. Genet Sel Evol, 33(2), 175–190.
Abstract: Genetic parameters of eventing horse competitions were estimated. About 13 000 horses, 30 000 annual results during 17 years and 110 000 starts in eventing competitions during 8 years were recorded. The measures of performance were logarithmic transformations of annual earnings, annual earnings per start, and annual earnings per place, and underlying variables responsible for ranks in each competition. Heritabilities were low (0.11 / 0.17 for annual results, 0.07 for ranks). Genetic correlations between criteria were high (greater than 0.90) except between ranks and earnings per place (0.58) or per start (0.67). Genetic correlations between ages (from 5 to 10 years old) were also high (more than 0.85) and allow selection on early performances. The genetic correlation between the results in different levels of competition (high/international and low/amateur) was near 1. Genetic correlations of eventing with other disciplines, which included partial aptitude needed for eventing, were very low for steeplechase races (0.18) and moderate with sport: jumping (0.45), dressage (0.58). The results suggest that selection on jumping performance will lead to some positive correlated response for eventing performance, but much more response could be obtained if a specific breeding objective and selection criteria were developed for eventing.
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Kruger, K., Gaillard, C., Stranzinger, G., & Rieder, S. (2005). Phylogenetic analysis and species allocation of individual equids using microsatellite data. Journal of Animal Breeding and Genetics, 122(s1), 78–86.
Abstract: Summary The taxonomic status of all equid species is not completely unravelled. This is of practical relevance for conservation initiatives of endangered, fragmented equid populations, such as the Asiatic wild asses (in particular Equus hemionus onager and E. hemionus kulan). In this study, a marker panel consisting of 31 microsatellite loci was used to assess species demarcation and phylogeny, as well as allocation of individuals (n = 120) to specific populations of origin (n = 11). Phylogenetic analysis revealed coalescence times comparable with those previously published from fossil records and mtDNA data. Using Bayesian approaches, it was possible to distinguish between the studied equids, although individual assignment levels varied. The observed results support the maintenance of separate captive conservation herds for E. hemionus onager and E. hemionus kulan. The first molecular genetic results for E. hemionus luteus remained contradictory, as they unexpectedly indicated a closer genetic relationship between E. hemionus luteus and E. kiang holderi compared with the other hemiones.
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Kristjansson, T., Bjornsdottir, S., Sigurdsson, A., Andersson, L. S., Lindgren, G., Helyar, S. J., et al. (2014). The effect of the ‘Gait keeper’ mutation in the DMRT3 gene on gaiting ability in Icelandic horses. J. Anim. Breed. Genet., , n/a-n/a.
Abstract: A nonsense mutation in DMRT3 (‘Gait keeper’ mutation) has a predominant effect on gaiting ability in horses, being permissive for the ability to perform lateral gaits and having a favourable effect on speed capacity in trot. The DMRT3 mutant allele (A) has been found in high frequency in gaited breeds and breeds bred for harness racing, while other horse breeds were homozygous for the wild-type allele (C). The aim of this study was to evaluate further the effect of the DMRT3 nonsense mutation on the gait quality and speed capacity in the multigaited Icelandic horse and demonstrate how the frequencies of the A- and C- alleles have changed in the Icelandic horse population in recent decades. It was confirmed that homozygosity for the DMRT3 nonsense mutation relates to the ability to pace. It further had a favourable effect on scores in breeding field tests for the lateral gait tölt, demonstrated by better beat quality, speed capacity and suppleness. Horses with the CA genotype had on the other hand significantly higher scores for walk, trot, canter and gallop, and they performed better beat and suspension in trot and gallop. These results indicate that the AA genotype reinforces the coordination of ipsilateral legs, with the subsequent negative effect on the synchronized movement of diagonal legs compared with the CA genotype. The frequency of the A-allele has increased in recent decades with a corresponding decrease in the frequency of the C-allele. The estimated frequency of the A-allele in the Icelandic horse population in 2012 was 0.94. Selective breeding for lateral gaits in the Icelandic horse population has apparently altered the frequency of DMRT3 genotypes with a predicted loss of the C-allele in relatively few years. The results have practical implications for breeding and training of Icelandic horses and other gaited horse breeds.
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Zhao, C. J., Qin, Y. H., Lee, X. H., & Wu, C. (2006). Molecular and cytogenetic paternity testing of a male offspring of a hinny. J Anim Breed Genet, 123(6), 403–405.
Abstract: An alleged male foal of a female mule, whose sire and grandparents were unknown, was identified for its pedigree. Parentage testing was conducted by comparing polymorphism of 12 microsatellite DNA sites and mitochondrial D-loop sequences of the male foal and the female mule. Both the sequence analysis of species-specific DNA fragments and a cytogenetic analysis were performed to identify the species of the foal and its parents. The results showed that the alleged female mule is actually a hinny, and the male foal, which possesses 62 chromosomes, qualifies as an offspring of the female hinny and a jack donkey.
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Berger J,. (1987). Reproductive fates of dispersers in a harem-dwelling ungulate: the wild horse. Mammalian dispersal Patterns, , 41–54.
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Robitaille, J., Brouillette, C., Lemieux, S., Perusse, L., Gaudet, D., & Vohl, M. C. (2004). Plasma concentrations of apolipoprotein B are modulated by a gene-diet interaction effect between the LFABP T94A polymorphism and dietary fat intake in French-Canadian men. Mol Genet Metabol, 82(4), 296–303.
Abstract: Hyperapobetalipoproteinemia is a common feature of the metabolic syndrome and could result from the interaction between genetic and dietary factors. The objective of this study was to verify whether dietary fat intake interacts with the T94A polymorphism of the liver fatty acid-binding protein (LFABP) gene to modulate plasma apolipoprotein (apo) B levels. Dietary fat and saturated fat intakes were obtained by a dietitian-administered food frequency questionnaire and the LFABP T94A genotype was determined by a PCR-RFLP based method in 623 French-Canadian men recruited through the Chicoutimi Lipid Clinic (279 T94/T94, 285 T94/A94, and 59 A94/A94). The LFABP T94A polymorphism was not associated with plasma apo B levels when fat intake was not taken into consideration. However, in a model including the polymorphism, fat intake expressed as a percentage of total energy intake, the interaction term and covariates, the variance in apo B concentrations was partly explained by the LFABP T94A polymorphism (5.24%, p=0.01) and by the LFABP T94A * fat interaction (6.25%, p=0.005). Results were similar when saturated fat replaced fat intake in the model (4.49%, p=0.02 for LFABP T94A and 6.43%, p=0.004 for the interaction). Moreover, in men consuming more than 30% of energy from fat, the odds ratio for having plasma apo B levels above 1.04 g/L for A94 carriers was of 0.40 (p=0.02) compared to T94/T94 homozygotes. Results were similar for carriers of the A94 allele consuming more than 10% of energy from saturated fat (OR: 0.32, p=0.005). In conclusion, T94/T94 exhibit higher apo B levels whereas carriers of the A94 allele seem to be protected against high apo B levels when consuming a high fat and saturated fat diet. These findings reinforce the importance to take into account gene-diet interactions in the prevention and management of the metabolic syndrome.
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Houpt, K., & Kusunose, R. (2000). Genetics of behaviour. In A. Ruvinsky A. T. Bowling (Ed.), The Genetics of the Horse (pp. 281–306). New York: CABI Publishing.
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