| Records |
| Author |
Hardy, J.L. |
| Title |
The ecology of western equine encephalomyelitis virus in the Central Valley of California, 1945-1985 |
Type |
Journal Article |
| Year |
1987 |
Publication |
The American Journal of Tropical Medicine and Hygiene |
Abbreviated Journal |
Am J Trop Med Hyg |
| Volume |
37 |
Issue |
3 Suppl |
Pages |
18s-32s |
Keywords  |
Aedes/microbiology; Animals; Birds; California; Culex/microbiology; Encephalitis Virus, Western Equine/*physiology; Encephalomyelitis, Equine/*history/microbiology/transmission/veterinary; History, 20th Century; Horse Diseases/history/transmission; Horses; Humans; Insect Vectors/microbiology; Mammals |
| Abstract |
Reeves' concept of the summer transmission cycle of western equine encephalomyelitis virus in 1945 was that the virus was amplified in a silent transmission cycle involving mosquitoes, domestic chickens, and possibly wild birds, from which it could be transmitted tangentially to and cause disease in human and equine populations. Extensive field and laboratory studies done since 1945 in the Central Valley of California have more clearly defined the specific invertebrate and vertebrate hosts involved in the basic virus transmission cycle, but the overall concept remains unchanged. The basic transmission cycle involves Culex tarsalis as the primary vector mosquito species and house finches and house sparrows as the primary amplifying hosts. Secondary amplifying hosts, upon which Cx. tarsalis frequently feeds, include other passerine species, chickens, and possibly pheasants in areas where they are abundant. Another transmission cycle that most likely is initiated from the Cx. tarsalis-wild bird cycle involves Aedes melanimon and the blacktail jackrabbit. Like humans and horses, California ground squirrels, western tree squirrels, and a few other wild mammal species become infected tangentially with the virus but do not contribute significantly to virus amplification. |
| Address |
Department of Biomedical and Environmental Health Sciences, School of Public Health, University of California, Berkeley 94720 |
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Thesis |
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Place of Publication |
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| Language |
English |
Summary Language |
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Original Title |
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Series Title |
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Series Issue |
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Edition |
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| ISSN |
0002-9637 |
ISBN |
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Expedition |
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| Notes |
PMID:3318522 |
Approved |
no |
| Call Number |
Equine Behaviour @ team @ |
Serial |
2677 |
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| Author |
Crans, W.J.; McNelly, J.; Schulze, T.L.; Main, A. |
| Title |
Isolation of eastern equine encephalitis virus from Aedes sollicitans during an epizootic in southern New Jersey |
Type |
Journal Article |
| Year |
1986 |
Publication |
Journal of the American Mosquito Control Association |
Abbreviated Journal |
J Am Mosq Control Assoc |
| Volume |
2 |
Issue |
1 |
Pages |
68-72 |
| Keywords |
Aedes/*microbiology; Alphavirus/*isolation & purification; Animals; Disease Outbreaks/veterinary; Ecology; Encephalitis Virus, Eastern Equine/*isolation & purification; Encephalomyelitis, Equine/epidemiology/transmission/veterinary; Horse Diseases/epidemiology/transmission; Horses; Insect Vectors/microbiology; New Jersey |
| Abstract |
Eastern equine encephalitis virus (EEE) was isolated from the salt marsh mosquito, Aedes sollicitans, collected from coastal areas of New Jersey on 3 occasions during the late summer and fall of 1982. The isolations were made at a time when local Culiseta melanura were either undergoing a population increase or exhibiting high levels of EEE virus. Although no human cases were reported during the epizootic period, the data lend support to the hypothesis that Ae. sollicitans is capable of functioning as an epidemic vector in the coastal areas of New Jersey where human cases of EEE have been most common. |
| Address |
Mosquito Research and Control, Cook College, Rutgers University, New Brunswick, NJ 08903 |
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Place of Publication |
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| Language |
English |
Summary Language |
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Original Title |
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Abbreviated Series Title |
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Series Issue |
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Edition |
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| ISSN |
8756-971X |
ISBN |
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Expedition |
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Conference |
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| Notes |
PMID:2853203 |
Approved |
no |
| Call Number |
Equine Behaviour @ team @ |
Serial |
2684 |
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| Author |
Ben-Shlomo, G.; Plummer, C.; Barrie, K.; Brooks, D. |
| Title |
Characterization of the normal dark adaptation curve of the horse |
Type |
Journal Article |
| Year |
2012 |
Publication |
Veterinary Ophthalmology |
Abbreviated Journal |
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| Volume |
15 |
Issue |
1 |
Pages |
42-45 |
| Keywords |
adaptation; curve; dark; electroretinography; equine; scotopic |
| Abstract |
Objective The goal of this work is to study the dark adaptation curve of the normal horse electroretinogram (ERG). Procedures The electroretinographic responses were recorded from six healthy female ponies using a contact lens electrode and a mini-Ganzfeld electroretinographic unit. The horses were sedated intravenously with detomidine, an auriculopalpebral nerve block was then performed, and the pupil was fully dilated. The ERG was recorded in response to a low intensity light stimulus (30 mcd.s/m2) that was given at times (T) T = 5, 10, 15, 20, 25, 30, 40, 50, and 60 min of dark adaptation. Off-line analysis of the ERG was then performed. Results Mean b-wave amplitude of the full-field ERG increased continuously from 5 to 25 min of dark adaptation. The b-wave amplitude peaked at T = 25, however, there was no statistical significance between T = 20 and T = 25. The b-wave amplitude then remained elevated with no significant changes until the end of the study at T = 60 (P > 0.49). The b-wave implicit time increased continuously between T = 5 and T = 20, then gradually decreased until T = 60. No distinct a-wave was observed during the testing time. Conclusions Evaluation of horse rod function or combined rod/cone function by means of full-field ERG should be performed after a minimum 20 min of dark adaptation. |
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| Publisher |
Blackwell Publishing Ltd |
Place of Publication |
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Series Issue |
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Edition |
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| ISSN |
1463-5224 |
ISBN |
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| Notes |
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Approved |
no |
| Call Number |
Equine Behaviour @ team @ |
Serial |
5626 |
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